Deflazacort Increases Laminin Expression and Myogenic Repair, and Induces Early Persistent Functional Gain in mdx Mouse Muscular Dystrophy
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چکیده
منابع مشابه
Persistent and improved functional gain in mdx dystrophic mice after treatment with L-arginine and deflazacort.
Although an increase in nitric oxide (NO) in muscle is reported to improve the outcome of deflazacort treatment for mdx mouse muscular dystrophy, the genetic homologue of Duchenne muscular dystrophy (DMD), the impact such treatment on the functional outcomes of the disease, including fiber susceptibility to exercise-induced injury, is not established. Experiments were designed to test whether t...
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ژورنال
عنوان ژورنال: Cell Transplantation
سال: 2000
ISSN: 0963-6897,1555-3892
DOI: 10.1177/096368970000900411